Sequestosome 1/p62 links familial ALS mutant SOD1 to LC3 via an ubiquitin-independent mechanism

The p62/sequestosome 1 protein has been identified as a component of pathological protein inclusions in neurodegenerative diseases including amyotrophic lateral sclerosis (ALS). P62 has also been implicated in autophagy, a process of mass degradation of intracellular proteins and organelles. Autopha...

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Bibliografski detalji
Glavni autori: Gal, Jozsef, Strom, Anna-Lena, Kwinter, David M., Kilty, Renee, Zhang, Jiayu, Shi, Ping, Fu, Weisi, Wooten, Marie W., Zhu, Haining
Format: Artigo
Jezik:Inglês
Izdano: 2009
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Article
Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC2766427/
https://ncbi.nlm.nih.gov/pubmed/19765191
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1471-4159.2009.06388.x
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