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Action potential clamp and chloroquine sensitivity of mutant Kir2.1 channels responsible for variant 3 short QT syndrome

Recently identified genetic forms of short QT syndrome (SQTS) are associated with an increased risk of arrhythmia and sudden death. The SQT3 variant is associated with an amino-acid substitution (D172N) in the KCNJ2-encoded Kir2.1 K(+) channel. In this study, whole-cell action potential (AP) clamp r...

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保存先:
書誌詳細
主要な著者: El Harchi, Aziza, McPate, Mark J., Zhang, Yi hong, Zhang, Henggui, Hancox, Jules C.
フォーマット: Artigo
言語:Inglês
出版事項: Academic Press 2009
主題:
オンライン・アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC2765655/
https://ncbi.nlm.nih.gov/pubmed/19285083
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2009.02.027
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