Mitochondrial dihydrolipoyl succinyltransferase deficiency accelerates amyloid pathology and memory deficit in a transgenic mouse model of amyloid deposition

Lignende værker

• Reduction of oxidative stress, amyloid deposition, and memory deficit by manganese superoxide dismutase overexpression in a transgenic mouse model of Alzheimer’s disease
  af: Dumont, Magali, et al.
  Udgivet: (2009)
• Mice deficient in dihydrolipoyl succinyltransferase show increased vulnerability to mitochondrial toxins
  af: Yang, Lichuan, et al.
  Udgivet: (2009)
• Coenzyme Q10 Decreases Amyloid Pathology and Improves Behavior in a Transgenic Mouse Model of Alzheimer’s Disease
  af: Dumont, Magali, et al.
  Udgivet: (2011)
• Apocynin administration does not improve behavioral and neuropathological deficits in a transgenic mouse model of Alzheimer’s disease
  af: Dumont, Magali, et al.
  Udgivet: (2011)
• PGC-1α overexpression exacerbates β-amyloid and tau deposition in a transgenic mouse model of Alzheimer's disease
  af: Dumont, Magali, et al.
  Udgivet: (2014)