Deficiency of NAD(P)H oxidase enhances hepatocellular injury but attenuates fibrosis after chronic carbon tetrachloride administration

Reactive oxygen species (ROS) activate hepatic stellate cells and enhance fibrogenesis. This study determine the role of NAD(P)H oxidase deficiency in the development of hepatocellular necrosis, inflammation and apoptosis in relation to fibrosis produced by chronic CCl(4) administration. Wild-type (...

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Autores principales: Aram, Ghazaleh, Potter, James J., Liu, Xiaopu, Wang, Lan, Torbenson, Michael S., Mezey, Esteban
Formato: Artigo
Lenguaje:Inglês
Publicado: 2009
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Article
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC2759707/
https://ncbi.nlm.nih.gov/pubmed/19072832
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/hep.22708
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