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Deficiency of NAD(P)H oxidase enhances hepatocellular injury but attenuates fibrosis after chronic carbon tetrachloride administration
Reactive oxygen species (ROS) activate hepatic stellate cells and enhance fibrogenesis. This study determine the role of NAD(P)H oxidase deficiency in the development of hepatocellular necrosis, inflammation and apoptosis in relation to fibrosis produced by chronic CCl(4) administration. Wild-type (...
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| Autors principals: | , , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2009
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2759707/ https://ncbi.nlm.nih.gov/pubmed/19072832 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/hep.22708 |
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