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XBP-1 deficiency in the nervous system protects against amyotrophic lateral sclerosis by increasing autophagy
Mutations in superoxide dismutase-1 (SOD1) cause familial amyotrophic lateral sclerosis (fALS). Recent evidence implicates adaptive responses to endoplasmic reticulum (ER) stress in the disease process via a pathway known as the unfolded protein response (UPR). Here, we investigated the contribution...
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| 主要な著者: | , , , , , , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Cold Spring Harbor Laboratory Press
2009
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2758741/ https://ncbi.nlm.nih.gov/pubmed/19762508 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1101/gad.1830709 |
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