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A BCR-ABL Mutant Lacking Direct Binding Sites for the GRB2, CBL and CRKL Adapter Proteins Fails to Induce Leukemia in Mice

The BCR-ABL tyrosine kinase is the defining feature of chronic myeloid leukemia (CML) and its kinase activity is required for induction of this disease. Current thinking holds that BCR-ABL forms a multi-protein complex that incorporates several substrates and adaptor proteins and is stabilized by mu...

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Bibliografiset tiedot
Päätekijät: Johnson, Kara J., Griswold, Ian J., O'Hare, Thomas, Corbin, Amie S., Loriaux, Marc, Deininger, Michael W., Druker, Brian J.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Public Library of Science 2009
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC2757918/
https://ncbi.nlm.nih.gov/pubmed/19823681
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0007439
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