Claudin-16 and claudin-19 interaction is required for their assembly into tight junctions and for renal reabsorption of magnesium

Claudins are tight junction integral membrane proteins that are key regulators of the paracellular pathway. Defects in claudin-16 (CLDN16) and CLDN19 function result in the inherited human renal disorder familial hypomagnesemia with hypercalciuria and nephrocalcinosis (FHHNC). Previous studies showe...

תיאור מלא

שמור ב:
מידע ביבליוגרפי
Main Authors: Hou, Jianghui, Renigunta, Aparna, Gomes, Antonio S., Hou, Mingli, Paul, David L., Waldegger, Siegfried, Goodenough, Daniel A.
פורמט: Artigo
שפה:Inglês
יצא לאור: National Academy of Sciences 2009
נושאים:
Biological Sciences
גישה מקוונת:https://ncbi.nlm.nih.gov/pmc/articles/PMC2741254/
https://ncbi.nlm.nih.gov/pubmed/19706394
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0907724106
תגים: הוספת תג
אין תגיות, היה/י הראשונ/ה לתייג את הרשומה!

פריטים דומים