Hsp27 inhibits sublethal, Src-mediated renal epithelial cell injury

Disruption of cell contact sites in renal epithelial cells contributes to organ dysfunction after ischemia. We hypothesized that heat shock protein 27 (Hsp27), a known cytoprotectant protein, preserves cell architecture and cell contact site function during ischemic stress. To test this hypothesis,...

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Bibliografiset tiedot
Päätekijät: Havasi, Andrea, Wang, Zhiyong, Gall, Jonathan M., Spaderna, Max, Suri, Vikram, Canlas, Ellery, Martin, Jody L., Schwartz, John H., Borkan, Steven C.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: American Physiological Society 2009
Aiheet:
Articles
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC2739706/
https://ncbi.nlm.nih.gov/pubmed/19553351
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.00052.2009
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