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Intracellular zinc inhibits KCC2 transporter activity

We report that KCC2 activity, monitored with wide–field fluorescence, is inhibited by intracellular Zn(2+), a key component of neuronal injury. Zn(2+)–mediated KCC2 inhibition produced a depolarizing shift of GABA(A) reversal potentials in rat neurons. Moreover, oxygen–glucose deprivation attenuated...

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Detalhes bibliográficos
Main Authors: Hershfinkel, Michal, Kandler, Karl, Knoch, Megan E., Dagan-Rabin, Michal, Aras, Mandar A., Abramovitch-Dahan, Chen, Sekler, Israel, Aizenman, Elias
Formato: Artigo
Idioma:Inglês
Publicado em: 2009
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2737597/
https://ncbi.nlm.nih.gov/pubmed/19430470
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/nn.2316
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