Suppression of Non-Homologous End Joining Repair by Overexpression of HMGA2

Understanding the molecular details associated with aberrant high mobility group A2 (HMGA2) gene expression is key to establishing the mechanism(s) underlying its oncogenic potential and impact on the development of therapeutic strategies. Here, we report the involvement of HMGA2 in impairing DNA-de...

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Detaylı Bibliyografya
Asıl Yazarlar: Li, Angela Y.J., Boo, Lee Ming, Wang, Shih-Ya, Lin, H. Helen, Wang, Clay C.C., Yen, Yun, Chen, Benjamin P.C., Chen, David J., Ann, David K.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: 2009
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Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC2737594/
https://ncbi.nlm.nih.gov/pubmed/19549901
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1158/0008-5472.CAN-08-4833
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