Mammalian Par3 regulates progenitor cell asymmetric division via Notch signaling in the developing neocortex

Asymmetric cell division of radial glial progenitors produces neurons while allowing self-renewal; however, little is known about the mechanism that generates asymmetry in daughter cell fate specification. Here we found that mammalian partition defective protein 3 (mPar3), a key cell polarity determ...

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Hlavní autoři: Bultje, Ronald S., Castaneda-Castellanos, David R., Jan, Lily Yeh, Jan, Yuh-Nung, Kriegstein, Arnold R., Shi, Song-Hai
Médium: Artigo
Jazyk:Inglês
Vydáno: 2009
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Article
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC2736606/
https://ncbi.nlm.nih.gov/pubmed/19640478
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neuron.2009.07.004
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