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Mammalian Par3 regulates progenitor cell asymmetric division via Notch signaling in the developing neocortex
Asymmetric cell division of radial glial progenitors produces neurons while allowing self-renewal; however, little is known about the mechanism that generates asymmetry in daughter cell fate specification. Here we found that mammalian partition defective protein 3 (mPar3), a key cell polarity determ...
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| Main Authors: | , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2009
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2736606/ https://ncbi.nlm.nih.gov/pubmed/19640478 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neuron.2009.07.004 |
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