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Novel N-terminal Cleavage of APP Precludes Aβ Generation in ACAT-Defective AC29 Cells

A common pathogenic event that occurs in all forms of Alzheimer’s disease is the progressive accumulation of amyloid β-peptide (Aβ) in brain regions responsible for higher cognitive functions. Inhibition of acyl-coenzyme A: cholesterol acyltransferase (ACAT), which generates intracellular cholestery...

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Detalhes bibliográficos
Main Authors: Huttunen, Henri J., Puglielli, Luigi, Ellis, Blake C., Ingano, Laura A. MacKenzie, Kovacs, Dora M.
Formato: Artigo
Idioma:Inglês
Publicado em: 2008
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2721794/
https://ncbi.nlm.nih.gov/pubmed/18618086
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s12031-008-9088-0
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