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Normal growth and development in mice over-expressing the CCN family member WISP3
Loss-of-function mutations in the gene WISP3 cause the autosomal recessive human skeletal disease Progressive Pseudorheumatoid Dysplasia, whereas mice with knockout mutations of Wisp3 have no phenotype. The lack of a phenotype in the Wisp3 knockout mice has constrained studies of the protein’s in vi...
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| Huvudupphovsmän: | , , , , |
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| Materialtyp: | Artigo |
| Språk: | Inglês |
| Publicerad: |
Springer Netherlands
2009
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| Ämnen: | |
| Länkar: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2721080/ https://ncbi.nlm.nih.gov/pubmed/19401829 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s12079-009-0040-z |
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