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Deficiency of the DNA repair enzyme ATM in rheumatoid arthritis

In rheumatoid arthritis (RA), dysfunctional T cells sustain chronic inflammatory immune responses in the synovium. Even unprimed T cells are under excessive replication pressure, suggesting an intrinsic defect in T cell regeneration. In naive CD4 CD45RA(+) T cells from RA patients, DNA damage load a...

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Detalles Bibliográficos
Main Authors: Shao, Lan, Fujii, Hiroshi, Colmegna, Inés, Oishi, Hisashi, Goronzy, Jörg J., Weyand, Cornelia M.
Formato: Artigo
Idioma:Inglês
Publicado: The Rockefeller University Press 2009
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Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC2715066/
https://ncbi.nlm.nih.gov/pubmed/19451263
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1084/jem.20082251
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