Kir6.2 Variant E23K Increases ATP-Sensitive K(+) Channel Activity and Is Associated With Impaired Insulin Release and Enhanced Insulin Sensitivity in Adults With Normal Glucose Tolerance

Lignende værker

• ATP inhibition of K(ATP) channels: control of nucleotide sensitivity by the N-terminal domain of the Kir6.2 subunit
  af: Koster, J C, et al.
  Udgivet: (1999)
• Sulfonylurea and K(+)-Channel Opener Sensitivity of K(ATP) Channels: Functional Coupling of Kir6.2 and Sur1 Subunits
  af: Koster, J.C., et al.
  Udgivet: (1999)
• Control of Rectification and Gating of Cloned K(ATP) Channels by the Kir6.2 Subunit
  af: Shyng, S.-L., et al.
  Udgivet: (1997)
• DEND Mutation in Kir6.2 (KCNJ11) Reveals a Flexible N-Terminal Region Critical for ATP-Sensing of the K(ATP) Channel
  af: Koster, Joseph C., et al.
  Udgivet: (2008)
• Engineered interaction between SUR1 and Kir6.2 that enhances ATP sensitivity in K(ATP) channels
  af: Pratt, Emily B., et al.
  Udgivet: (2012)