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Chk2 is required for HSV-1 ICP0-mediated G2/M arrest and enhancement of virus growth
ICP0 is a multi-functional herpes simplex virus type 1 (HSV-1) immediate-early (IE) gene product that contributes to efficient virus growth and reactivation from latency. Here we show that HSV-1-induced cell-cycle arrest at the G2/M border requires ICP0 and Chk2 kinase and that ICP0 expression by tr...
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| Autors principals: | , , , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2008
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2706573/ https://ncbi.nlm.nih.gov/pubmed/18321553 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.virol.2008.01.038 |
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