Transcription of the protein kinase C-δ gene is activated by JNK through c-Jun and ATF2 in response to the anticancer agent doxorubicin

Lignende værker

• Enzyme Kinetics and Interaction Studies for Human JNK1β1 and Substrates Activating Transcription Factor 2 (ATF2) and c-Jun N-terminal kinase (c-Jun)
  af: Figuera-Losada, Mariana, et al.
  Udgivet: (2012)
• Reovirus Infection Activates JNK and the JNK-Dependent Transcription Factor c-Jun
  af: Clarke, Penny, et al.
  Udgivet: (2001)
• ATF-2 is preferentially activated by stress-activated protein kinases to mediate c-jun induction in response to genotoxic agents.
  af: van Dam, H, et al.
  Udgivet: (1995)
• Doxorubicin Requires the Sequential Activation of Caspase-2, Protein Kinase Cδ, and c-Jun NH(2)-terminal Kinase to Induce Apoptosis
  af: Panaretakis, Theocharis, et al.
  Udgivet: (2005)
• Phosphorylation of ATF2 and Interaction with NFY Induces c-Jun in the Gonadotrope
  af: Lindaman, Lacey L., et al.
  Udgivet: (2012)