טוען...
Loss of Podocyte aPKCλ/ι Causes Polarity Defects and Nephrotic Syndrome
Atypical protein kinase C (aPKC) is a central component of the evolutionarily conserved Par3-Par6-aPKC complex, one of the fundamental regulators of cell polarity. We recently demonstrated that these proteins interact with Neph-nephrin molecules at the slit diaphragm of the glomerular filtration bar...
שמור ב:
| Main Authors: | , , , , , , , , |
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| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
American Society of Nephrology
2009
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2663834/ https://ncbi.nlm.nih.gov/pubmed/19279126 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1681/ASN.2008080871 |
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