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Distinct roles of stress-activated protein kinases in Fanconi anemia type C–deficient hematopoiesis
The underlying molecular mechanisms that promote bone marrow failure in Fanconi anemia are incompletely understood. Evidence suggests that enhanced apoptosis of hematopoietic precursors is a major contributing factor. Previously, enhanced apoptosis of Fanconi anemia type C–deficient (Fancc(−/−)) pro...
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| Autors principals: | , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
American Society of Hematology
2009
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2661855/ https://ncbi.nlm.nih.gov/pubmed/19168785 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1182/blood-2008-09-181420 |
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