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Dual DNA unwinding activities of the Rothmund–Thomson syndrome protein, RECQ4
Human RECQ helicases have been linked to distinct clinical diseases with increased cancer rates and premature ageing. All RECQ proteins, except RECQ4, have been shown to be functional helicases. Mutations in RECQ4 lead to Rothmund–Thomson syndrome (RTS), and mouse models reveal that the conserved he...
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| Hoofdauteurs: | , |
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| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
Nature Publishing Group
2009
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2657580/ https://ncbi.nlm.nih.gov/pubmed/19177149 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/emboj.2009.13 |
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