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Hyperoxia-induced premature senescence requires p53 and pRb, but not mitochondrial matrix ROS
Senescence is a potential tumor-suppressing mechanism and a commonly used model of cellular aging. One current hypothesis to explain senescence, based in part on the correlation of oxygen with senescence, postulates that it is caused by oxidative damage from reactive oxygen species (ROS). Here, we f...
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| Hlavní autoři: | , , , , , , , , |
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| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
The Federation of American Societies for Experimental Biology
2009
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2653981/ https://ncbi.nlm.nih.gov/pubmed/18948382 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1096/fj.08-114256 |
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