The Ψ(m) depolarization that accompanies mitochondrial Ca(2+) uptake is greater in mutant SOD1 than in wild-type mouse motor terminals

Lignende værker

• Repetitive Nerve Stimulation Transiently Opens the Mitochondrial Permeability Transition Pore in Motor Nerve Terminals of Symptomatic Mutant SOD1 Mice
  af: Nguyen, Khanh T., et al.
  Udgivet: (2011)
• Extrusion of Ca(2+) from mouse motor terminal mitochondria via a Na(+)–Ca(2+) exchanger increases post-tetanic evoked release
  af: García-Chacón, Luis E, et al.
  Udgivet: (2006)
• Stimulation-Evoked Increases in Cytosolic [Ca(2+)] in Mouse Motor Nerve Terminals Are Limited by Mitochondrial Uptake and Are Temperature-Dependent
  af: David, Gavriel, et al.
  Udgivet: (2000)
• Early vulnerability to ischemia/reperfusion injury in motor terminals innervating fast muscles of SOD1-G93A mice
  af: David, Gavriel, et al.
  Udgivet: (2007)
• Mitochondrial Ca(2+) uptake prevents desynchronization of quantal release and minimizes depletion during repetitive stimulation of mouse motor nerve terminals
  af: David, Gavriel, et al.
  Udgivet: (2003)