DNA damage and p53-mediated cell cycle arrest: A reevaluation
Most mammalian cells exhibit transient delays in the G(1) and G(2) phases of the cell cycle after treatment with radiation or radiomimetic compounds. p53 is required for the arrest in G(1), which provides time for DNA repair. Recently, a role of p53 in the G(2)/M transition has also been suggested....
Uloženo v:
| Vydáno v: | Proc Natl Acad Sci U S A |
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| Hlavní autoři: | , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
National Academy of Sciences
1996
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC26382/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8986789/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.93.26.15209 |
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