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High glucose-induced IKK-Hsp-90 interaction contributes to endothelial dysfunction
A decline in the bioavailability of nitric oxide (NO) that causes endothelial dysfunction is a hallmark of diabetes. The availability of NO to the vasculature is regulated by endothelial nitric oxide synthase (eNOS) activity and the involvement of heat shock protein-90 (Hsp-90) in the regulation of...
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| Main Authors: | , , , , |
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| Format: | Artigo |
| Jezik: | Inglês |
| Izdano: |
American Physiological Society
2009
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| Teme: | |
| Online dostop: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2636992/ https://ncbi.nlm.nih.gov/pubmed/18945937 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpcell.00575.2007 |
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