Transgenic Mice Expressing Dominant Negative Bright Exhibit Defects in B1 B Cells

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• The Transcription Factor Bright Plays a Role in Marginal Zone B Lymphocyte Development and Autoantibody Production
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• Loss of Bright/ARID3a Function Promotes Developmental Plasticity
  per: An, Guangyu, et al.
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• Anti-Nuclear Antibody Production and Autoimmunity in Transgenic Mice that Over-Express the Transcription Factor Bright
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• Defective Erythropoiesis in Transgenic Mice Expressing Dominant-Negative Upstream Stimulatory Factor
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• Bruton's Tyrosine Kinase Regulates Immunoglobulin Promoter Activation in Association with the Transcription Factor Bright
  per: Rajaiya, Jaya, et al.
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