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Loss of Bright/ARID3a Function Promotes Developmental Plasticity
Bright (B cell regulator of immunoglobulin heavy chain transcription)/ARID3a, an A+T-rich interaction domain protein, was originally discovered in B lymphocyte lineage cells. However, expression patterns and high lethality levels in knockout mice suggested it had additional functions. Three independ...
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| Главные авторы: | , , , , , , |
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| Формат: | Artigo |
| Язык: | Inglês |
| Опубликовано: |
2010
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| Предметы: | |
| Online-ссылка: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2977942/ https://ncbi.nlm.nih.gov/pubmed/20680960 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/stem.491 |
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