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The N-terminal domain of a K(+) channel β subunit increases the rate of C-type inactivation from the cytoplasmic side of the channel

Voltage-gated K(+) channels are complexes of membrane-bound, ion-conducting α and cytoplasmic ancillary (β) subunits. The primary physiologic effect of coexpression of α and β subunits is to increase the intrinsic rate of inactivation of the α subunit. For one β subunit, Kvβ1.1, inactivation is enha...

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Main Authors: Morales, Michael J., Wee, Jonny O., Wang, Shimin, Strauss, Harold C., Rasmusson, Randall L.
Formato: Artigo
Idioma:Inglês
Publicado: The National Academy of Sciences of the USA 1996
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Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC26366/
https://ncbi.nlm.nih.gov/pubmed/9005448
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