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Co-crystal structures of primed side-extending α-ketoamide inhibitors reveal novel calpain-inhibitor aromatic interactions

Calpains are intracellular cysteine proteases that catalyze the cleavage of target proteins in response to Ca(2+) signaling. When Ca(2+) homeostasis is disrupted, calpain over-activation causes unregulated proteolysis, which can contribute to diseases such as post-ischemic injury and cataract format...

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Détails bibliographiques
Auteurs principaux: Qian, Jin, Cuerrier, Dominic, Davies, Peter L., Li, Zhaozhao, Powers, James C., Campbell, Robert L.
Format: Artigo
Langue:Inglês
Publié: 2008
Sujets:
Accès en ligne:https://ncbi.nlm.nih.gov/pmc/articles/PMC2631982/
https://ncbi.nlm.nih.gov/pubmed/18702462
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1021/jm800045t
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