Absence of the complement regulatory molecule CD59a leads to exacerbated neuropathology after traumatic brain injury in mice

BACKGROUND: Complement represents a crucial mediator of neuroinflammation and neurodegeneration after traumatic brain injury. The role of the terminal complement activation pathway, leading to generation of the membrane attack complex (MAC), has not been thoroughly investigated. CD59 is the major re...

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Detalhes bibliográficos
Main Authors: Stahel, Philip F, Flierl, Michael A, Morgan, B Paul, Persigehl, Ivonne, Stoll, Christiane, Conrad, Claudia, Touban, Basel M, Smith, Wade R, Beauchamp, Kathryn, Schmidt, Oliver I, Ertel, Wolfgang, Leinhase, Iris
Formato: Artigo
Idioma:Inglês
Publicado em: BioMed Central 2009
Assuntos:
Research
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2631471/
https://ncbi.nlm.nih.gov/pubmed/19133139
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1742-2094-6-2
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