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Macrophage EP4 deficiency increases apoptosis and suppresses early atherosclerosis

Prostagladin (PG) E(2), a major product of activated macrophages, has been implicated in atherosclerosis and plaque rupture. The PGE(2) receptors, EP2 and EP4, are expressed in atherosclerotic lesions and are known to inhibit apoptosis in cancer cells. To examine the roles of macrophage EP4 and EP2...

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Bibliographische Detailangaben
Hauptverfasser:	Babaev, Vladimir R., Chew, Joshua D., Ding, Lei, Davis, Sarah, Breyer, Matthew D., Breyer, Richard M., Oates, John A., Fazio, Sergio, Linton, MacRae F.
Format:	Artigo
Sprache:	Inglês
Veröffentlicht:	2008
Schlagworte:	Article
Online Zugang:	https://ncbi.nlm.nih.gov/pmc/articles/PMC2614698/ https://ncbi.nlm.nih.gov/pubmed/19041765 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.cmet.2008.09.005
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Macrophage EP4 deficiency increases apoptosis and suppresses early atherosclerosis

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