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IL-17A and IL-17F do not contribute vitally to autoimmune neuro-inflammation in mice

The clear association of Th17 cells with autoimmune pathogenicity implicates Th17 cytokines as critical mediators of chronic autoimmune diseases such as EAE. To study the impact of IL-17A on CNS inflammation, we generated transgenic mice in which high levels of expression of IL-17A could be initiate...

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Bibliografiset tiedot
Päätekijät: Haak, Stefan, Croxford, Andrew L., Kreymborg, Katharina, Heppner, Frank L., Pouly, Sandrine, Becher, Burkhard, Waisman, Ari
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: American Society for Clinical Investigation 2008
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC2613466/
https://ncbi.nlm.nih.gov/pubmed/19075395
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI35997
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