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IL-17A and IL-17F do not contribute vitally to autoimmune neuro-inflammation in mice
The clear association of Th17 cells with autoimmune pathogenicity implicates Th17 cytokines as critical mediators of chronic autoimmune diseases such as EAE. To study the impact of IL-17A on CNS inflammation, we generated transgenic mice in which high levels of expression of IL-17A could be initiate...
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| Hoofdauteurs: | , , , , , , |
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| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
American Society for Clinical Investigation
2008
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2613466/ https://ncbi.nlm.nih.gov/pubmed/19075395 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI35997 |
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