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Salt and acid-base metabolism in claudin-16 knockdown mice: impact for the pathophysiology of FHHNC patients
Claudin-16 is defective in familial hypomagnesemia with hypercalciuria and nephrocalcinosis (FHHNC). Claudin-16 knockdown (CLDN16 KD) mice show reduced cation selectivity in the thick ascending limb. The defect leads to a collapse of the lumen-positive diffusion voltage, which drives Ca(2+) and Mg(2...
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| Main Authors: | , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
American Physiological Society
2008
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2604832/ https://ncbi.nlm.nih.gov/pubmed/18784260 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.90388.2008 |
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