ロード中...

Disruption of Survivin in K562 cells elevates telomerase activity and protects cells against apoptosis induced by the Bcr-abl kinase inhibitor STI571

The Bcr-abl kinase inhibitor STI571 produces clinical responses in most patients with Chronic Myeloid Leukemia (CML); however, development of resistance limits utility. One strategy to overcome STI571 resistance is to decrease the level/activity of Bcr-abl. We reported that disruption of the anti-ap...

詳細記述

保存先:
書誌詳細
主要な著者: Wang, Zhanxiang, Pelus, Louis M.
フォーマット: Artigo
言語:Inglês
出版事項: 2008
主題:
オンライン・アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC2600525/
https://ncbi.nlm.nih.gov/pubmed/19079792
タグ: タグ追加
タグなし, このレコードへの初めてのタグを付けませんか!