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Heme oxygenase-1 deficiency leads to disrupted response to acute stress in stem cells and progenitors
An effective response to extreme hematopoietic stress requires an extreme elevation in hematopoiesis and preservation of hematopoietic stem cells (HSCs). These diametrically opposed processes are likely to be regulated by genes that mediate cellular adaptation to physiologic stress. Herein, we show...
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| Main Authors: | , , , , , , , , |
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| Formáid: | Artigo |
| Teanga: | Inglês |
| Foilsithe: |
American Society of Hematology
2008
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| Ábhair: | |
| Rochtain Ar Líne: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2597124/ https://ncbi.nlm.nih.gov/pubmed/18509090 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1182/blood-2007-12-127621 |
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