Increased intracellular Ca(2+) and SR Ca(2+) load contribute to arrhythmias after acidosis in rat heart. Role of Ca(2+)/calmodulin-dependent protein kinase II

Returning to normal pH after acidosis, similar to reperfusion after ischemia, is prone to arrhythmias. The type and mechanisms of these arrhythmias have never been explored and were the aim of the present work. Langendorff-perfused rat/mice hearts and rat-isolated myocytes were subjected to respirat...

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Main Authors: Said, M., Becerra, R., Palomeque, J., Rinaldi, G., Kaetzel, M. A., Diaz-Sylvester, P. L., Copello, J. A., Dedman, J. R., Mundiña-Weilenmann, C., Vittone, L., Mattiazzi, A.
Formato: Artigo
Idioma:Inglês
Publicado em: American Physiological Society 2008
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Articles
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2593495/
https://ncbi.nlm.nih.gov/pubmed/18723772
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00010.2008
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