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Contribution of the B oligomer to the protective activity of genetically attenuated pertussis toxin.

An enzymatically deficient recombinant S1 subunit, in which Arg-9 was replaced by Lys, was combined with native B oligomer to form a mutant holotoxin molecule. This molecule exhibited decreased leukocytosis-promoting and histamine-sensitizing activities compared with those of the native toxin, suppo...

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Autores principales: Arciniega, J L, Shahin, R D, Burnette, W N, Bartley, T D, Whiteley, D W, Mar, V L, Burns, D L
Formato: Artigo
Lenguaje:Inglês
Publicado: 1991
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Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC258899/
https://ncbi.nlm.nih.gov/pubmed/1894354
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