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Activation of Hsp90-eNOS and increased NO generation attenuate respiration of hypoxia-treated endothelial cells
Hypoxia induces various adoptive signaling in cells that can cause several physiological changes. In the present work, we have observed that exposure of bovine aortic endothelial cells (BAECs) to extreme hypoxia (1–5% O(2)) attenuates cellular respiration by a mechanism involving heat shock protein...
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| Päätekijät: | , , , , |
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| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
American Physiological Society
2008
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2585002/ https://ncbi.nlm.nih.gov/pubmed/18787079 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpcell.00550.2007 |
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