Platelet-activating factor modulates endotoxin-induced macrophage procoagulant activity by a protein kinase C-dependent mechanism.
Macrophage procoagulant activity is an important mediator of extravascular fibrin deposition at sites of infection and appears to contribute to the pathogenesis of several infectious disease processes. Previous studies have shown that the inflammatory mediator platelet-activating factor was able to...
Tallennettuna:
| Julkaisussa: | Infect Immun |
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| Päätekijät: | , , |
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
American Society for Microbiology (ASM)
1992
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC257578/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/1541568/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/iai.60.3.944-950.1992 |
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