Platelet-activating factor modulates endotoxin-induced macrophage procoagulant activity by a protein kinase C-dependent mechanism.

Macrophage procoagulant activity is an important mediator of extravascular fibrin deposition at sites of infection and appears to contribute to the pathogenesis of several infectious disease processes. Previous studies have shown that the inflammatory mediator platelet-activating factor was able to...

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Опубликовано в::	Infect Immun
Главные авторы:	Kucey, D S, Cheung, P Y, Rotstein, O D
Формат:	Artigo
Язык:	Inglês
Опубликовано:	American Society for Microbiology (ASM) 1992
Предметы:	Research Article
Online-ссылка:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC257578/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/1541568/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/iai.60.3.944-950.1992
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