Human Papillomavirus Type 16 E7 Oncoprotein Can Induce Abnormal Centrosome Duplication through a Mechanism Independent of Inactivation of Retinoblastoma Protein Family Members

Documenti analoghi

• Centrosome Abnormalities and Genomic Instability by Episomal Expression of Human Papillomavirus Type 16 in Raft Cultures of Human Keratinocytes
  di: Duensing, Stefan, et al.
  Pubblicazione: (2001)
• Human Papillomavirus Type 16 E7 Oncoprotein Associates with the Centrosomal Component γ-Tubulin
  di: Nguyen, Christine L., et al.
  Pubblicazione: (2007)
• Degradation of the Retinoblastoma Tumor Suppressor by the Human Papillomavirus Type 16 E7 Oncoprotein Is Important for Functional Inactivation and Is Separable from Proteasomal Degradation of E7
  di: Gonzalez, Sonia L., et al.
  Pubblicazione: (2001)
• The human papillomavirus type 16 E6 and E7 oncoproteins cooperate to induce mitotic defects and genomic instability by uncoupling centrosome duplication from the cell division cycle
  di: Duensing, Stefan, et al.
  Pubblicazione: (2000)
• The Human Papillomavirus Type 16 E6 Oncoprotein Activates mTORC1 Signaling and Increases Protein Synthesis
  di: Spangle, Jennifer M., et al.
  Pubblicazione: (2010)