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Cisplatin Cytotoxicity of Auditory Cells Requires Secretions of Proinflammatory Cytokines via Activation of ERK and NF-κB

The ototoxicity of cisplatin, a widely used chemotherapeutic agent, involves a number of mechanisms, including perturbation of redox status, increase in lipid peroxidation, and formation of DNA adducts. In this study, we demonstrate that cisplatin increased the early immediate release and de novo sy...

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Main Authors:	So, Hongseob, Kim, HyungJin, Lee, Jeong-Han, Park, Channy, Kim, Yunha, Kim, Eunsook, Kim, Jin-Kyung, Yun, Ki-Jung, Lee, Kang-Min, Lee, Haa-Yung, Moon, Sung-Kyun, Lim, David J., Park, Raekil
Format:	Artigo
Jezik:	Inglês
Izdano:	Springer-Verlag 2007
Teme:	Article
Online dostop:	https://ncbi.nlm.nih.gov/pmc/articles/PMC2538433/ https://ncbi.nlm.nih.gov/pubmed/17516123 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s10162-007-0084-9
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Cisplatin Cytotoxicity of Auditory Cells Requires Secretions of Proinflammatory Cytokines via Activation of ERK and NF-κB

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