Influenza virus regulates protein synthesis during infection by repressing autophosphorylation and activity of the cellular 68,000-Mr protein kinase.

We investigated the mechanisms by which influenza virus prevents shutoff of protein synthesis by a cellular protein kinase normally activated during infection. Earlier work has shown that influenza virus superinfection of cells previously infected by the adenovirus VAI RNA-negative mutant dl331 resu...

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Библиографические подробности

Опубликовано в::	J Virol
Главные авторы:	Katze, M G, Tomita, J, Black, T, Krug, R M, Safer, B, Hovanessian, A
Формат:	Artigo
Язык:	Inglês
Опубликовано:	American Society for Microbiology (ASM) 1988
Предметы:	Research Article
Online-ссылка:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC253514/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/3418783/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jvi.62.10.3710-3717.1988
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