Influenza virus regulates protein synthesis during infection by repressing autophosphorylation and activity of the cellular 68,000-Mr protein kinase.

Podobné jednotky

• The cellular 68,000-Mr protein kinase is highly autophosphorylated and activated yet significantly degraded during poliovirus infection: implications for translational regulation.
  Autor: Black, T L, a další
  Vydáno: (1989)
• Purification and partial characterization of a cellular inhibitor of the interferon-induced protein kinase of Mr 68,000 from influenza virus-infected cells.
  Autor: Lee, T G, a další
  Vydáno: (1990)
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  Autor: Katze, M G, a další
  Vydáno: (1987)
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• The integrity of the stem structure of human immunodeficiency virus type 1 Tat-responsive sequence of RNA is required for interaction with the interferon-induced 68,000-Mr protein kinase.
  Autor: Roy, S, a další
  Vydáno: (1991)