Ability of a T-antigen transport-defective mutant of simian virus 40 to immortalize primary cells and to complement polyomavirus middle T in tumorigenesis.

Eitemau Tebyg

• Sequences from polyomavirus and simian virus 40 large T genes capable of immortalizing primary rat embryo fibroblasts.
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  Cyhoeddwyd: (1985)
• Polyomavirus large T mutants affected in retinoblastoma protein binding are defective in immortalization.
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  Cyhoeddwyd: (1991)
• Mutation in the polyomavirus genome that activates the properties of large T associated with neoplastic transformation.
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  Cyhoeddwyd: (1986)
• Complementation Analysis of Simian Virus 40 Mutants
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  Cyhoeddwyd: (1974)
• Functional complementation of nuclear targeting-defective mutants of simian virus 40 structural proteins.
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  Cyhoeddwyd: (1994)