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Pathological angiogenesis is induced by sustained Akt signaling and inhibited by rapamycin

Endothelial cells in growing tumors express activated Akt, which when modeled by transgenic endothelial expression of myrAkt1 was sufficient to recapitulate the abnormal structural and functional features of tumor blood vessels in nontumor tissues. Sustained endothelial Akt activation caused increas...

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Detalhes bibliográficos
Main Authors: Phung, Thuy L., Ziv, Keren, Dabydeen, Donnette, Eyiah-Mensah, Godfred, Riveros, Marcela, Perruzzi, Carole, Sun, Jingfang, Monahan-Earley, Rita A., Shiojima, Ichiro, Nagy, Janice A., Lin, Michelle I., Walsh, Kenneth, Dvorak, Ann M., Briscoe, David M., Neeman, Michal, Sessa, William C., Dvorak, Harold F., Benjamin, Laura E.
Formato: Artigo
Idioma:Inglês
Publicado em: 2006
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2531257/
https://ncbi.nlm.nih.gov/pubmed/16904613
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.ccr.2006.07.003
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