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RPA phosphorylation facilitates mitotic exit in response to mitotic DNA damage
Human replication protein A (RPA) becomes phosphorylated on the RPA2 subunit by cyclin B-Cdc2 during mitosis, although the functional role of this modification is unclear. We find that this modification stimulates RPA2 to become hyperphosphorylated in response to mitotic DNA damage caused by bleomyc...
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| 主要な著者: | , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
National Academy of Sciences
2008
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| 主題: | |
| オンライン・アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2529075/ https://ncbi.nlm.nih.gov/pubmed/18723675 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0803001105 |
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