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Latent infections in spinal ganglia with thymidine kinase-deficient herpes simplex virus.

A herpes simplex virus type 1 variant [C239(TK-)] harboring a deletion in the thymidine kinase (TK) gene was assessed for capacity to establish latent infections. Outbred Swiss Webster mice were inoculated on both hind footpads, and numbers of neurons expressing latency-associated transcript and amo...

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Detalles Bibliográficos
Publicado en:J Virol
Autores principales: Leist, T P, Sandri-Goldin, R M, Stevens, J G
Formato: Artigo
Lenguaje:Inglês
Publicado: American Society for Microbiology (ASM) 1989
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Acceso en línea:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC251150/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2552180/
https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jvi.63.11.4976-4978.1989
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